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UID:1002@biology.technion.ac.il

DTSTART;TZID=Asia/Jerusalem:20211012T130000

DTEND;TZID=Asia/Jerusalem:20211012T140000

DTSTAMP:20211118T110055Z

URL:https://biology.technion.ac.il/en/seminars/graduate-msc-seminar-noa-sc
 hneider-2/

SUMMARY:Graduate MSC Seminar -Noa Schneider [No Categories]
DESCRIPTION:Location:   \n Affiliation: \n Host:\n Abstract:RNA editing is 
 a highly conserved mechanism by which theAdenosine Deaminases Acting on RN
 A (ADAR) enzymes convert adenosines (A) intoinosines (I)\, which are recog
 nized by the splicing and translationalmachineries as guanosines (G). This
  seemingly small modification has anextensive effect both on the structure
  and function of the edited RNA. RNAediting has been found to be involved 
 in viral RNA defense\, mRNA recoding\, andneuronal diseases. Because RNA e
 diting is important for neuronal function\,ADARs are essential in mammals.
  However\, in Caenorhabditis elegans\,deletion of either or both of its AD
 AR genes is not lethal\, making thiswell-characterized model organism idea
 l for this kind of research.  In my work I studied the localization of A
 DR-2\, thecatalytically active ADAR in C. elegans\, how this localization 
 isregulated and how it affects RNA editing activity. Also\, I found differ
 ences intarget selection between human ADAR2 and C. elegans ADR-2. Exploit
 ingthese differences\, I built a system to predict human ADAR2 target sele
 ction.   

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