BEGIN:VCALENDAR

VERSION:2.0

PRODID:-//wp-events-plugin.com//7.2.2.1//EN

TZID:Asia/Jerusalem

X-WR-TIMEZONE:Asia/Jerusalem
BEGIN:VEVENT

UID:1197@biology.technion.ac.il

DTSTART;TZID=Asia/Jerusalem:20230904T130000

DTEND;TZID=Asia/Jerusalem:20230904T133000

DTSTAMP:20230829T084133Z

URL:https://biology.technion.ac.il/en/seminars/msc-graduate-seminar-galit-
 cohen/

SUMMARY:Msc Graduate Seminar- Galit Cohen [No Categories]
DESCRIPTION:Location: Faculty Of Biology Auditorium/ZOOM:https://technion.z
 oom.us/j/99587628620    Galit Cohen\n Affiliation: \n Host:Associate Prof.
   Shenhav Cohen   \n Developing a nutrient-based formulation to attenuate 
 muscle wasting\nMuscle atrophy causes weakness and disability in aging\, i
 nactivity\, and in many diseases. This loss of muscle mass and strength is
  primarily due to the accelerated degradation of muscle proteins by the ub
 iquitin-proteasome system (UPS). The ubiquitin ligases\, F-Box (MAFbx/Atro
 gin-1) and Muscle RING-Finger 1 (MuRF-1)\, are induced in most types of at
 rophy and currently represent the best markers for atrophy. Their expressi
 on increases also by the Transforming Growth Factor-β (TGF-β) family mem
 ber\, myostatin\, which functions as a negative regulator of skeletal musc
 le mass. Myostatin is produced by muscle\, and is excessively active in sy
 stemic catabolic conditions (including cancer cachexia and aging)\, causin
 g a severe muscle atrophy. Its inhibition is currently considered as the b
 est therapeutic approach to prevent or retard atrophy. Here we focus on th
 e development of a nutrient-based formulation that specifically targets my
 ostatin signaling for the treatment of muscle wasting in humans. 
LOCATION:Faculty Of Biology Auditorium/ZOOM:https://technion.zoom.us/j/9958
 7628620 

END:VEVENT

BEGIN:VTIMEZONE

TZID:Asia/Jerusalem

X-LIC-LOCATION:Asia/Jerusalem

BEGIN:DAYLIGHT

DTSTART:20230324T030000

TZOFFSETFROM:+0200

TZOFFSETTO:+0300

TZNAME:IDT

END:DAYLIGHT

END:VTIMEZONE
END:VCALENDAR